Wind turbine study timing curious

by J. Morris, Ingersoll Times
The Ontario government’s December 16 press release relates to a report commissioned by the Ministry of the Environment and written by HGC, an engineering firm. The timing of this press release is curious, as it appears the same report was in draft form in August 2010 then submitted as a Final Draft December 2010 by the MOE as evidence during the Environmental Review Tribunal and finally to the public in August 2011. Why would it suddenly warrant a press release in December 2011? Could it be an attempt to divert attention from the upcoming appeal hearing in the Watford area for the Zephyr wind project?

Statements about ‘no direct health effects’ are a red herring, because it is acknowledged by international experts (including experts called upon by the MOE and wind energy proponents) that wind turbines cause adverse health effects via the ‘indirect’ pathway. Even Mr. Howe (of HGC) acknowledged under oath at the 2011 Environmental Review Tribunal (ERT) that indirect effects are still ‘something that can occur’.

The press release raises a number of questions:

– When it comes to health, why is the government commissioning reports and relying on information generated by an engineering firm, who is a member of the Canadian Wind Energy Association?

– Why are desk top literature reviews still the government’s method of choice to address concerns rather than properly designed independent field epidemiology studies performed by qualified epidemiology experts?

– Why was the press release issued now, when the referenced report was compiled in 2010?

The press release and report unfortunately do no justice to the families who have already been impacted and displace from their homes at projects throughout the province due to adverse health effects.

It is important to reiterate that, after weeks of testimony from experts around the world in 2011, the Environmental Review Tribunal for the Kent Breezes project concluded:

“This case has successfully shown that the debate should not be simplified to one about whether wind turbines can cause harm to humans. The evidence presented to the tribunal demonstrated that they can, if facilities are placed too close to residents. The debate has now evolved to one of degree.”

J. Morris, Woodstock

5 thoughts on “Wind turbine study timing curious

  1. Our reps and servants at work to protect themselves and big corps and there own hides. throw out the diversion card,now please, Screw the bosses (that’s us if your forgot).

  2. Plus the recent McPherson study indicated that infrasound may be able to have a direct impact upon health. It can be found at




    Is this government doing all it can to cover up a major mistake rather than giving a proper provable study.?

    Is it saying that the Liberal Government’s survival goes ahead of the health and welfare of Ontario People. If they are safe, why are they not in cities? Why is Premier McGuinjty not boasting of having one on his street in Ottawa or on Queens Park?

    We ask you, “Are the uncountable numbers of people living within 2 miles of turbines all around the world lying when they complain that newly installed turbines cause disorientation, nausea, vertigo, high blood pressure sessions, increased respiratory rates, loss of balance, intolerable sensations, headaches, dizziness incapacitation, disorientation, incapacitation, heart palpitations, sleep disturbances, fatigue, apathy depression, loss of concentration, apathy, increased diastolic and decreased systolic and high or low blood pressure, altered time perception, and changes in behavior.?

    Is the work of countless scientists and medical people wrong when they cite and explain clearly how Vibroacoustic disease VAD produced by these wind turbines cause permanent deamage as in thi report from the renouned
    ERISA, Lusofona University, Avenida Primeiro de Maio, No. 27, 5B, Costa da Caparica, 2825 397 Lisbon, Portugal.

    Abstract The damage from infra-sound is serious, permanent and is not just some momentary discomfort.
    Vibroacoustic disease (VAD) is a whole-body pathology that develops in individuals excessively exposed to ILFN. VAD has been diagnosed within several professional groups employed within the aeronautical industry, and in other heavy industries. However, given the ubiquitous nature of ILFN and the absence of legislation concerning ILFN, VAD is increasingly being diagnosed among members of the general population, including children. VAD is associated with the abnormal growth of extra-cellular matrices (collagen and elastin), in the absence of an inflammatory process. In VAD, the end-product of collagen and elastin growth is reinforcement of structural integrity. This is seen in blood vessels, cardiac structures, trachea, lung, and kidney of both VAD patients and ILFN-exposed animals. VAD is, essentially, a mechanotransduction disease. Inter- and intra-cellular communication is achieved through both biochemical and mechanotranduction signalling.
    [That is both physical damage like getting a punch and through how these vibrations alter the functions of the endocrine and nervous systems In other words, if we are chased by a bear, the adrenalin increase s .infra sound does the
    When the structural components of tissue are altered, as is seen in ILFN-exposed specimens, the mechanically mediated signalling is, at best, impaired. Common medical diagnostic tests, such as EKG, EEG, as well as many blood chemistry analyses, are based on the mal-function of biochemical signalling processes. VAD patients typically present normal values for these tests. However, when echocardiography, brain MRI or histological studies are performed, where structural changes can be identified, all consistently show significant changes in VAD patients and ILFN-exposed animals. Frequency-specific effects are not yet known, valid dose-responses have been difficult to identify, and large-scale epidemiological studies are still lacking.

    How ultra sound damages our bodies

    These documentations explain that while we do not sense It because it is outside of our hearing range = infra sound causes our body sensitive organ parts to vibrate. It is somewhat being chased by a tiger without end and how this would upset our hormones
    What happens when it makes our organs vibrate?

    Infrasound would seem to cause PERMANENT Blood vessel thickening, cochlear damage, reduced physical stamina, damage to liver and other organs, and especially the endocrine system and nervous system, both of which work together to control all functions of our bodied
    nd these documentations explain that while we do not sense It = infra sound apparently causes our body sensitive organ parts to vibrate
    What happens when it makes our organs vibrate?
    • In July of 2002, Ising and Ising published a study that discusses how noise, perceived as a threat, stimulates release of cortisol.


    • The infra sound apparently causes the body organs to vibrate. People are unaware. But the result is serious disruptions
    Cortisol (hydrocortisone) is a steroid hormone, more specifically a glucocorticoid, produced by the adrenal gland. It is released in response to stress. Its primary functions are to increase blood sugar through gluconeogenesis; suppress the immune system; and aid in fat, protein and carbohydrate metabolism.[1] It also decreases bone formation
    Cortisol is released in response to stress, acting to restore homeostasis. However, prolonged cortisol secretion (which may be due to chronic stress or the excessive secretion seen in Cushing’s syndrome) FROM INFRA SOUND AND SO WIND TURBINES results in significant physiological changes in human bodies
    Cortisol counteracts insulin, contributes to hyperglycemia-causing hepatic gluconeogenesis and inhibits the peripheral utilization of glucose (insulin resistance) ] by decreasing the translocation of glucose transporters (especially GLUT4) to the cell membranes ] However, cortisol increases glycogen synthesis (glycogenesis) in the liver. The permissive effect of cortisol on insulin action in liver glycogenesis is observed in hepatocyte culture in the laboratory, although the mechanism for this is unknown.

    In laboratory rats, cortisol-induced collagen loss in the skin is ten times greater than in any other tissue. Cortisol (as opticortinol) may inversely inhibit IgA precursor cells in the intestines of calves.[17] Cortisol also inhibits IgA in serum, as it does IgM; however, it is not shown to inhibit IgE.[18]
    Gastric and renal secretion
    Cortisol stimulates gastric-acid secretion. Cortisol’s only direct effect on the hydrogen ion excretion of the kidneys is to stimulate the excretion of ammonium ions by deactivating the renal glutaminase enzyme. Net chloride secretion in the intestines is inversely decreased by cortisol in vitro (methylprednisolone).[21]
    Cortisol inhibits sodium loss through the small intestine of mammals. Sodium depletion, however, does not affect cortisol levels so cortisol cannot be used to regulate serum sodium. Cortisol’s original purpose may have been sodium transport. This hypothesis is supported by the fact that freshwater fish utilize cortisol to stimulate sodium inward, while saltwater fish have a cortisol-based system for expelling excess sodium.

    A sodium load augments the intense potassium excretion by cortisol; corticosterone is comparable to cortisol in this case.[25] In order for potassium to move out of the cell, cortisol moves an equal number of sodium ions into the cell.[26] This should make pH regulation much easier (unlike the normal potassium-deficiency situation, in which two sodium ions move in for each three potassium ions that move out—closer to the deoxycorticosterone effect). Nevertheless, cortisol consistently causes serum alkalosis; in a deficiency, serum pH does not change. The purpose of this may be to reduce serum pH to an optimum value for some immune enzymes during infection, when cortisol declines. Potassium is also blocked from loss in the kidneys by a decline in cortisol (9 alpha fluorohydrocortisone)
    Cortisol acts as a diuretic hormone, controlling one-half of intestinal diuresis; it has also been shown to control kidney diuresis in dogs. The decline in water excretion following a decline in cortisol (dexamethasone) in dogs is probably due to inverse stimulation of antidiuretic hormone (ADH or arginine vasopressin), which is not overridden by water loading. Humans and other animals also use this mechanism.
    Cortisol stimulates many copper enzymes (often to 50% of their total potential), probably to increase copper availability for immune purposes.This includes lysyl oxidase, an enzyme which is used to cross-link collagen and elastin Especially valuable for immune response is cortisol’s stimulation of the superoxide dismutase, since this copper enzyme is almost certainly used by the body to permit superoxides to poison bacteria. Cortisol causes an inverse four- or fivefold decrease of metallothionein (a copper storage protein) in mice;[33] however, rodents do not synthesize cortisol themselves. This may be to furnish more copper for ceruloplasmin synthesis or to release free copper. Cortisol has an opposite effect on aminoisobuteric acid than on the other amino acids. If alpha-aminoisobuteric acid is used to transport copper through the cell wall, this anomaly might be explained.

    Immune system
    Cortisol can weaken the activity of the immune system. Cortisol prevents proliferation of T-cells by rendering the interleukin-2 producer T-cells unresponsive to interleukin-1 (IL-1), and unable to produce the T-cell growth factor.[35] Cortisol also has a negative-feedback effect on interleukin-1. IL-1 must be especially useful in combating some diseases; however, endotoxic bacteria have gained an advantage by forcing the hypothalamus to increase cortisol levels (forcing the secretion of CRH hormone, thus antagonizing IL-1). The suppressor cells are not affected by glucosteroid response-modifying factor (GRMF),so the effective setpoint for the immune cells may be even higher than the setpoint for physiological processes (reflecting leukocyte redistribution to lymph nodes, bone marrow, and skin). Rapid administration of corticosterone (the endogenous Type I and Type II receptor agonist) or RU28362 (a specific Type II receptor agonist) to adrenalectomized animals induced changes in leukocyte distribution. Natural killer cells are not affected by cortisol]
    Bone metabolism
    Cortisol reduces bone formation, favoring long-term development of osteoporosis. It transports potassium out of cells in exchange for an equal number of sodium ions (see above).[39] This can trigger the hyperkalemia of metabolic shock from surgery. Cortisol also reduces calcium absorption in the intestine.]
    Cortisol works with epinephrine (adrenaline) to create memories of short-term emotional events; this is the proposed mechanism for storage of flash bulb memories, and may originate as a means to remember what to avoid in the future. However, long-term exposure to cortisol damages cells in the hippocampus; this damage results in impaired learning. Furthermore, it has been shown that cortisol inhibits memory retrieval of already stored information
    Additional effects
    • Increases blood pressure by increasing the sensitivity of the vasculature to epinephrine and norepinephrine; in the absence of cortisol, widespread vasodilation occurs.
    • Inhibits secretion of corticotropin-releasing hormone (CRH), resulting in feedback inhibition of ACTH (Adrenocorticotropic hormone or corticotropin) secretion. Some researchers believe that this normal feedback system may become dysregulated when animals are exposed to chronic stress.
    • Causes the kidneys to produce hypotonic urine
    • Shuts down the reproductive system, resulting in an increased chance of miscarriage and (in some cases) temporary infertility. Fertility returns after cortisol levels return to norma;
    \Has anti-inflammatory properties, reducing histamine secretion and stabilizing lysosomal membranes. Stabilization of lysosomal membranes prevents their rupture, preventing damage to healthy tissues.
    • Stimulates hepatic detoxification by inducing tryptophan oxygenase (reducing serotonin levels in the brain), glutamine synthase (reducing glutamate and ammonia levels in the brain), cytochrome P-450 hemoprotein (mobilizing arachidonic acid), and metallothionein (reducing heavy metals in the body)
    • In addition to cortisol’s effects in binding to the glucocorticoid receptor, because of its molecular similarity to aldosterone it also binds to the mineralocorticoid receptor. Aldosterone and cortisol have a similar affinity for the mineralocorticoid receptor; however, glucocorticoids circulate at roughly 100 times the level of mineralocorticoids. An enzyme exists in mineralocorticoid target tissues to prevent overstimulation by glucocorticoids and allow selective mineralocorticoid action. This enzyme—11-beta hydroxysteroid dehydrogenase type II (Protein:HSD11B2)—catalyzes the deactivation of glucocorticoids to 11-dehydro metabolitesd
    • There are potential links between cortisol, appetite and obesity.WORLD RENOWNED EXPERT ISSUES DIRE WARNINGS TO RESIDENTS OF ONTARIO
    Picton, Ontario, January 15, 2009
    200 Picton area residents braved frigid temperatures Thursday night to hear about the serious health risks associated with Industrial Wind Turbines.
    Dr. Magda Havas Ph.D., Associate Professor of Environmental and Resource Studies at Peterborough’s Trent University and one of the world’s leading research experts in the areas of electromagnetic pollution, radio frequency radiation, ground current and dirty electricity delivered alarming warnings about the dangers to human and animal health, posed by Industrial Wind Turbines.
    Dr. Havas was the featured speaker in Picton. Havas identified two significant problem areas that pose substantive threats to the health of those living or working in proximity to wind turbine developments – Infrasound or Low Frequency Sound emitted by the turbines themselves and both Dirty Electricity and stray voltage related to the electricity produced by turbines.
    The human body feels and reacts to sound waves that we cannot hear. These waves, in the low end of the sound spectrum (under 20 Hz), are well documented to cause nausea, joint pain, insomnia, depression, agitation, increased blood pressure and possible heart problems. It is also a major cause of Vibroacoustic Disease which can lead to vision problems, digestive problems, cardiovascular problems, and circulatory difficulties.
    Since infrasound can carry over very long distances, Dr. Havas says that the jury is still out on how far away from people Industrial Wind Turbines should be located. She quoted the French Academy of Medicine guidelines which call for a minimum setback of 1.5 kilometers and Dr. Nina Pierpont of New York State, who has done comprehensive research into turbine related illnesses, who calls for a minimum of 2 kilometers.
    But Dr. Havas was quick to point out that infrasound is not the only danger to local residents posed by wind turbines. She is particularly concerned about the impacts of “Dirty Electricity” also a byproduct of Wind Turbines in Ontario, “… the worst part of it is that it could all be eliminated with proper design and care”, Havas said. “However, there is a cost involved….do you think that they will spend the money necessary to correct a problem that they refuse to admit even exists?”
    Some of the symptoms created by Dirty Electricity in the home resemble those caused by infrasound. They include nausea, sleep disorders, headaches, dizziness, tinnitus and irregular heart beat. Havas has done important research into this area and studies have identified substantially increased rates of cancers, increased blood sugar among some diabetics, skin sensitivities, and other equally disturbing conditions.
    Havas’ presentation followed that of Bill Palmer a Professional Engineer with experience in risk assessment and public safety. Palmer identified the fact that Hydro One has setback guidelines to protect their critical Hydro lines from possible turbine damage but the Ontario Government has put no setbacks in place to protect people. “Hydro requires a setback of 500 meters from their most important lines – to protect against mechanical damage from a turbine collapse or blade failure”, Palmer said. “Unfortunately, no such guidelines are in place for public safety to protect people traveling on roadways, in their fields adjacent to a turbine, or in their homes…..There are no guidelines in place to protect citizens from these same mechanical risks that Hydro One has setbacks for. He went on to show how the calculation of the setback required for noise from wind turbines needed to take into consideration the change in wind profile at night, the cyclic nature of wind turbine noise, and the low frequency component of wind turbine noise.”.
    This also apples to power lines from wind turbines.
    Moderator, Dr. Robert McMurtry, founding Assistant Deputy Minister of the Population and Public Health Branch of Health Canada and special advisor to Roy Romanow during the Romanow Commission, expressed concern about the lack of public protection in place. He agreed with Palmer and Havas that Industrial Wind Turbine developments must be controlled and that the current Public Health Guidelines concerning setback distances must be reassessed. Even the recommended setback distances of 2km may not be appropriate – epidemiological studies are necessary to set the record straight.
    Dr. Robert McMurtry M.D. F.R.C.S(C), F.A.C.S. Professor Emeritus of Surgery at the University of Western Ontario spoke about the need for an epidemiological study into the health impacts of industrial wind turbines and the inadequacy of current setback guidelines with regards to people. Adverse health effects are being noted by people in Ontario who live near wind turbine installations. Dr. McMurtry has been gathering data from health specialists around the world who are documenting medical evidence of physiological adverse effects of the noise from wind turbines. Many case reports and case series have emerged but no authoritative epidemiological studies. As the Ministry of Environment (Ontario) regulations are inadequate such a study is imperative. Dr. McMurtry also pointed out the resolution of the French Academy of Scientists and the Darmstadt Manifesto from over 100 scientists in Germany regarding set-backs.
    Mr. William Palmer a Professional Engineer discussed the need for provincial standards to be established to protect the public. Palmer noted that in the case of Hydro One Networks Inc. a distance of 500 meters from wind power developments has been set in order to protect their electricity transmission lines. However there is no requirement as to how far away wind installations must be from roads and lot lines. He cited cases where wind installations in Ontario are as near as 50 meters from non participating neighbor’s lot lines. It was then pointed out that this is the recommended municipal bylaw information from CanWea, the Canadian Wind Energy Association.
    Mr. Palmer also discussed evidence collected over two years to show the change in wind profile that makes wind turbines particularly annoying at night, and described why the cyclic noise produced by wind turbines makes them more annoying than a steady sound.

    There are many other studies and reports that show the dangers associated with wind turbines and infra sound



  4. It may interest you all to learn that like chemical and biological weapons, many nations of the world have banned infrasonic weapons as well. This was done many decades ago.
    Go here for confirmation of the sum of your infrasonic fears:
    Some rather ominous damage to various organs including the brain have been documented. The list of research labs includes the who’s who of academia.


    Sean Holt.

Leave a Reply

Your email address will not be published. Required fields are marked *